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Birnbaum RS, Bertelsen AH and Roos BA 1989 ; Glucocorticoid regulation of amidating enzyme in a neoplastic C-cell line. Mol Cell Endocrinol 61: 109 116. Bradbury AF and Smyth DG 1991 ; Peptide amidation. Trends Biochem Sci 16: 112 115. Eipper BA, Green CB-R, Campbell TA, Stoffers DA, Keutmann HT, Mains RE and Ouafik LH 1992b ; Alternative splicing and endoproteolytic processing generate tissue-specific forms of pituitary peptidylglycine -amidating monooxygenase PAM ; . J Biol Chem 267: 4008 4015. Eipper BA, Meyers AC and Mains RE 1985 ; Peptidyl-glycine amidating activity in tissues and serum of the adult rat. Endocrinology 116: 24972504. Eipper BA, Milgram SL, Husten EJ, Yun H-Y and Mains RE 1993 ; Peptidylglycine -amidating monooxygenase: a multifunctional protein with catalytic, processing, and routing domains. Protein Science 2: 489 497. Eipper BA, Quon AS, Mains RE, Boswell JS and Blackburn NJ 1995 ; The catalytic core of peptidylglycine alpha-hydroxylating monooxygenase: Investigation by sitedirected mutagenesis, Cu X-ray absorption spectroscopy, and electron paramagnetic resonance. Biochemistry 34: 28572865. Eipper BA, Stoffers DA and Mains RE 1992a ; The biosynthesis of neuropeptides: Peptide -amidation. Annu Rev Neurosci 15: 57 85. Faiman MD 1987 ; Disulfiram: Pharmacologic, pharmacokinetic and toxicologic considerations, in Disulfiram Antabuse ; a Unique Medical Aid to Sobriety McNichol RW, Ewing JA and Faiman MD, eds ; pp 19 46, Charles C Thomas, Springfield, IL. Grino M, Guillaume V, Boudouresque F, Conte-Devoix B, Maltese J-Y and Oliver C 1990 ; Glucocorticoids regulate peptidylglycine -amidating monooxygenase gene expression in the rat hypothalamic paraventricular nucleus. Mol Endocrinol 4: 16131619. Gronemeyer H 1992 ; Control of transcription activation by steroid hormone receptors. FASEB J 6: 2524 2529. Hand TA, Mains RE and Eipper BA 1996 ; Identification of the promoter for the gene encoding the bifunctional enzyme, peptidylglycine -amidating monooxygenase. DNA Cell Biol 15: 10931104. Hart DW and Faiman MD 1992 ; In vitro and in vivo inhibition of rat liver aldehyde dehydrogenase by S-methyl-N, N-diethylthiolcarbamate sulfoxide, a new metabolite of disulfiram. Biochem Pharmacol 43: 403 406. Husten EJ, Tausk FA, Keutmann HT and Eipper BA 1993 ; Use of endoproteases to identify catalytic domains, linker regions, and functional interactions in soluble peptidylglycine -amidating monooxygenase. J Biol Chem 268: 9709 9717. Kaler SG, Goldstein DS, Holmes C, Salerno JA and Gahl WA 1993 ; Plasma and cerebrospinal fluid neurochemical patterns in Menkes disease. Ann Neurol 33: 171175. Kapuscinski M, Green M, Sinha SN, Shepard JJ and Shulkes A 1993 ; Peptide -amidation activity in human plasma: Relationship to gastrin processing. Clin Endocrinol Oxf ; 39: 551558. Katopodis AG and May SW 1990 ; Novel substrates and inhibitors of peptidylglycine -amidating monooxygenase. Biochemistry 29: 4541 4548. Laemmli UK 1970 ; Cleavage of structural proteins during the assembly of the head of bacteriophage T4. Nature 227: 680 685. Lowry OH, Rosebrough NJ, Farr AL and Randall RJ 1951 ; Protein measurement with the Folin phenol reagent. J Biol Chem 193: 265275. Mains RE, Meyers AC and Eipper BA 1985 ; Hormonal, drug, and dietary factors affecting peptidylglycine -amidating monooxygenase activity in various tissues of the adult male rat. Endocrinology 116: 25052515. Maltese J-Y and Eipper BA 1992 ; Developmental expression of peptidylglycine -amidating monooxygenase PAM ; in primary cultures of neonatal rat cardiocytes: A model for studying regulation of PAM expression in the rat heart. Mol Endocrinol l6: 1998 2008. Maltese J-Y, Oyarce and Eipper BA 1996 ; Prevalence and turnover of peptidyl.
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Healthy older sister. She has been told that she will receive an immunosuppressive regimen that will permit the total withdrawal of corticosteroids within a few days of the transplant. She is concerned about the safety of this regimen and seeks your advice. Which ONE of the following statements regarding transplant immunosuppression is correct? A. She should agree to total withdrawal of steroids only if she is part of a randomized clinical trial. B. It would be safer to withdraw the steroids after several months rather than a few days after the transplant. C. Early steroid withdrawal protocols have been shown to reduce the risk for diabetes compared with regimens that maintain low-dosage steroids. D. At many transplant centers, early steroid withdrawal protocols are now routine in patients who are deemed to be at low immunologic risk. E. Early steroid withdrawal protocols require higher maintenance dosages of calcineurin inhibitors. 21. A 48-yr-old Caucasian male physician with ESRD as a result of IgA nephropathy is scheduled to receive a living-donor transplant from his one-haplotype-matched healthy older sister. He questions you regarding the use of antibody induction immunosuppression. Which ONE of the following statements is true? A. For patients at high immunologic risk, there may be a lower risk for acute rejection with Thymoglobulin use compared with the IL-2 receptor antagonists. B. For patients at low immunologic risk, there is no advantage to using antibody induction. C. Antibody induction is typically used only for high-risk patients in the United States. D. Randomized, prospective clinical trials have shown that alemtuzumab Campath.
Our lives are stressful. We know that. Relationships, career, family, education, and striving toward personal fulfillment can all contribute to feelings of tension and anxiety. Most people realize that we live in a culture full of stressors and are well aware of the challenge to juggle a number of responsibilities. This works in our favor because when we're aware of what causes stress in our lives, we find that we are better able to deal with it. But with so many stressors that we know about, is it possible that we may be stressed out by things we don't even realize are there? Yes. As women we are faced with a number of stressors that operate on a deeper, more discreet level. There are expectations and pressures forced upon us by our culture that we may not even be conscious of. While sexism can still be blatant, it often takes place in more subtle ways. As a result, we may not notice its existence, but still feel its effects. It may occur in the workplace or the classroom causing us to question our abilities or feel frustrated for not being recognized for the abilities we have. It may occur at home as we feel unidentified pressure to take care of a larger percentage of the household and childcare responsibilities. Without even noticing it, we may take on more than our share and then wonder why we're so exhausted by the end of the day. We are also faced with pressure from the media to fit a physically perfect and unattainable ideal. We may spend excessive amounts of time and energy focused on.
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Amoxicillin, cefaclor, cephalexin, and trimethoprim-sulfamethoxazole. J Infect Dis. 1988; 158: 474-477. Anne S, Reisman RE. Risk of administering cephalosporin antibiotics to patients with histories of penicillin allergy. Ann Allergy Asthma Immunol. 1995; 74: 167-170. Arndt KA, Jick H. Rates of cutaneous reactions to drugs: a report from the Boston Collaborative Drug Surveillance Program. JAMA. 1976; 235: 918-923. Shapiro S, Slone D, Siskind V, Lewis GP, Jick H. Drug rash with ampicillin and other penicillins. Lancet. 1969; 2: 969-972. Kraemer MJ, Smith AL. Rashes with ampicillin. Pediatr Rev. 1990; 1: 197-201. Koch-Weser J, Sidel VW, Dexter M, Parish C, Finer DC, Kanarek P. Adverse reactions to sulfisoxazole, sulfamethoxazole, and nitrofurantoin. Arch Intern Med. 1971; 128: 399-404. Roujeau J-C, Kelly JP, Naldi L, et al. Medication use and the risk of StevensJohnson syndrome or toxic epidermal necrolysis. N Engl J Med. 1995; 333: 16001607. Uhari M, Nuutinen M, Turtinen J. Adverse reactions in children during longterm antimicrobial therapy. Pediatr Infect Dis J. 1996; 15: 404-408. Pichichero ME, Pichichero DM. Diagnosis of penicillin, amoxicillin, and cephalosporin allergy: reliability of examination assessed by skin testing and oral challenge. J Pediatr. 1998; 132: 137-143. Worrall GJ, Hull C, Briffett E. Radioallergosorbent testing for penicillin allergy in family practice. CMAJ. 1994; 150: 37-41. Anderson JA. Cross-sensitivity to cephalosporins in patients allergic to penicillin. Pediatr Infect Dis. 1986; 5: 557-561. DeLeo VA. Skin testing in systemic cutaneous drug reactions. Lancet. 1998; 352: 1488-1490. Matsui D, McLauchlin M, Rieder MJ. Predictive value of oral sulfonamide challenge for adverse reactions. Pediatr Infect Dis J. 1997; 16: 1984-1985.
Abbreviations: CARE, Cholesterol and Recurrent Events study; EXCEL, Expanded Clinical Evaluation of Lovastatin trial; KAPS, Kuopio Atherosclerosis Prevention Study; LIPID, Long-term Intervention With Pravastatin in Ischemic Disease trial; LIPS, Lescol Intervention Prevention Study; MIRACL, myocardial ischemia reduction with aggressive cholesterol lowering; PPP, Prospective Pravastatin Pooling project; PROSPER, Prospective Study of Pravastatin in the Elderly at Risk; WOSCOP, West of Scotland Coronary Prevention study. * Only studies with comments on relevance of subjective adverse effects are included and antara.
To the site of angioplasty in either the carotid or iliac artery and immediately inflated at 4 atm of pressure for 5 minutes. All balloon dilations were performed with a balloon to artery ratio of approximately 1.3: 1 as determined by intravascular ultrasound. Longer inflations were not used because of the potential for ischemia when long inflation durations are performed for clinical angioplasty. All animal studies conforned to guiding principles of the American Physiological Society.
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The body requires a constant supply of glucose to maintain normal function. Known hypoglycemic patients need glucose levels restored as soon as possible to reduce brain and other organ damage. Hypoglycemia is a life-threatening problem. The prehospital goal is to maintain stable vital signs, protect the patient's airway and C-spine, and assess for possible causes. Get as complete a history as possible. Restore glucose levels as soon as possible and antispasmodic.
Bray, H. G., Carpanini, F. M. B., and Waters, B. D. 1971 ; . The metabolism of thiophene in the rabbit and rat Xenobiotica 1, 157-168. Brennan, L. M., and Creasia, D. A. 1982 ; . The effects of methapyrilene hydrochloride on hepatocarcinogenicity and pentobarbitaJ-induced sleeping time in rats and mice. Toxicol. Appl. Pharmacol. 66, 252-258. Burke, M. D., and Mayer, R. T. 1983 ; . Differential effects of phenobarbitone and 3-methylcholanthrene induction on the hepatic microsomal metabolism and cytochrome P-450-binding of phenoxanzone and a homologous series of n-octyl ethers alkoxyresorufins ; . Chem. Biol. Interact. 45, 243-258. Cottrell, S., Oliver, K., Lake, B. G., and Powell, C. J. 1996 ; . Strain-specific enhancement or inhibition of coumarin hepatotoxicity in mice following pretreatment with two different liver enzyme-inducing agents. Fundam, Appl. Toxicol. 34, 47-55. Dalvi, R. R., and Dalvi, P. S. 1991 ; . Differences in the effects of piperine and piperonyl butoxide on hepatic drug-metabolising system in rats. Drug. Chem. Toxicol. 14, 219-229. Dansette, P. M., Amar, C , Smith, C , Pons, C., and Mansuy, D. 1990 ; . Oxidative activation of the thiophene ring by hepatic enzymes. Biochem. Pharmacol. 39, 911-918. Dansette, P. M., Thang, D. C , El Amri, H., and Mansuy, D. 1992 ; . Evidence for thiophene-S-oxide as a primary reactive metabolite of thiophene in vivo: Formation of a dihydrothiophene sulfoxide mercapturic acid. Biochem. Biophys. Res. Commun. 186, 1624-1630. Drummond, G. S., and Kappas, A. 1982 ; . The cytochrome P450 depleted animal: An experimental model for in vivo studies in chemical biology. Proc. Natl. Acad. Sci. USA 79, 2384-2388. Graichen, M. E., Neptun, D. A., Dent, J. G., Popp, J. A., and Leonard, T. B. 1985 ; . Effects of methapyrilene on rat hepauc xenobioUc enzymes and liver morphology. Fundam. Appl. Toxicol. 5, 165-174. Jiang, X-M., Cantrill, E., Farrell, G. C , and Murray, M. 1994 ; . Pretranslational down-regulation of male specific hepatic P450s after portal bypass. Biochem. Pharmacol. 48, 701-708. Kammerer, R. C., and Schmitz, D. A. 1987 ; . Species differences in the in vitro metabolism of methapyrilene. Xenobiotica 17, 1121-1130. Kammerer, R. C , and Schmitz, D. A. 1986 ; . Metabolism of methapyrilene by rat-liver homogenate. Xenobiotica 16, 671-680. Kammerer, R. C , Schmitz, D. A., Lampe, M. A., and Kloc, K. 1988 ; . The in vivo metabolism of methapyrilene, a hepatocarcinogen, in the rat. Xenobiotica 18, 869-881. Kelly, D. W., Holder, C. L., Korfmacher, W. A., Getek, T. A., Lay, J. O. Jr., Casciano, D. A., Shaddock, J. G., Duhart, H. M., and Slikker Jr., W. 1992 ; . Metabolism of methapyrilene by Fischer-344 rat and B6C3F, mouse hepatocytes. Xenobiotica 22, 1367-1381.
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